Liver Disorders Quiz part 1
By:Mohamed Mamdouh
Liver Disorders Quiz
Test your knowledge on liver disorders with our comprehensive quiz designed for learners at all levels. Dive into a series of questions that explore liver fun
Whether you’re a student or a professional, this quiz will help you clarify key concepts related to liver health.
- 52 challenging questions
- Instant feedback on your answers
- Improves knowledge about liver disorders
High vascular organ located behind the ribs in the upper right portions
Pancreas
Liver
Lymph
Bladder
80% of liver blood supply rich in nutrients but lack of oxygen comes from
Hepatic artery
Radial artery
Portal vein
Arch of aorta
20% of liver blood supply and rich in oxyegn comes from
Radial artery
Hepatic artery
Portal vein
Arch of aorta
Formation of glycogen from glucose
Glycogenolysis
Gluconeogenesis
Fat Metabolism
Glycogenesis
Breakdown of glycogen to glucose
Glycogenesis
Glycogenolysis
Gluconeogenesis
Fat Metabolism
The synthesis of glucose from certain amino acids, lactate or glycerol
Glycogenesis
Glycogenolysis
Gluconeogenesis
Fat Metabolism
Results in formation of ammonia and considered a toxin
Glycogenesis
Glycogenolysis
Gluconeogenesis
Fat Metabolism
The liver synthesizes all of the plasma proteins Except
Alpha globulin
Gamma globulin
Beta globulin
Prothrombin
Is required by the liver for synthesis of prothrombin
Vitamin A
Vitamin B
Vitamin D
Vitamin K
Fat metabolism is production of
Energy
Ketones
Cholesterol
All of the above
The liver can be storage for
Vitamins A K E D
Iron and copper
B12
All of the above
Stored in gallbladder and composed mainly of water and electrolytes
Bilirubin
Bile
Cholesterol
Protein
Derived from breakdown of old hemoglobin and removed from the liver
Bile
Protein
Bilirubin
Vitamins
Excreted by the kidneys in the urine and came from bilirubin
Protein
Vitamins
Urobilinogen
Hemoglobin
Ingest bacteria or other foreign material from the blood
Beta cells
Kupffer cells
Alpha cells
Gamma cells
Which of the following is function of liver
A) drug metabolism
B) protective function
C) iron metabolism
Both A,B
Aid to digestion through emulsification of fats
Urobilinogen
Barbiturates
Bile salts
Opioids
Liver may be palpable in the
Left upper quadrant
Left lower quadrant
Right upper quadrant
Right lower quadrant
Normal level of serum bilirubin
A) 0-0.3-1.2 mg/dL
B) 0-5.1 mol/L
C) 1.7-20.5 mol/dL
Both A,B
Total serum protein normal value
A) 7.0-7.5 g/dL
B) 4.0-5.5 g/dL
C) 1.7-3.3 g/dL
Both A ,B
Serum albumin normal value
A) 7.0-7.5 g/dL
B) 4.0-5.5 g/dL
C) 1.7-3.3 g/dL
Both A ,C
Serum globulin normal value
A) 7.0-7.5 g/dL
B) 4.0-5.5 g/dL
C) 1.7-3.3 g/dL
0-0.3 mg/dL
Prothrombin normal value
A)100%
B)12-16 seconds
C)80% -20-25
Only C
Both A.B
Can be preformed with ultrasound guidance or transvenously through right internal jugular vein to right hepatic vein
Liver biopsy
MRI
Laparoscopy
Paracentesis
May be preformed to assess liver size and hepatic blood flow and obstruction using chemicals
MRI
CT
Radioisotope liver
Ultrasound
Hemolytic jaundice is considered .......... disease
Pre hepatic
Hepatic
Post hepatic
Non hepatic
Which of the following is the cause of hemolytic jaundice
A) transfusion reaction
B) hepatits viruses
C) pigment stones in gallbladder
Both A,C
Hemolytic jaundice is the result of
Increased destruction of the red blood cells
Normal destruction of the red blood cells
Alcohol intake
Chemical toxins
Extremely severe jaundice level is
0.3-1.3 mg/dL
0-0.3 mg/dL
20-25 mg/dL
None of the above
The liver in hemolytic jaundice is
Semi-functioning
Non-functioning
Destroyed
Functioning normally
Jaundice becomes clinically evident when the serum bilirubin level is
0-0.3mg/dL
0.3-1.3 mg/dL
> 2.5 mg/dL
All of the above
Hepatocellular jaundice is considered .......... disease
Pre hepatic
Hepatic
Post hepatic
Non hepatic
Caused by the inability of damaged liver cells to clear normal amounts of bilirubin from blood
Hemolytic jaundice
Obstructive jaundice
Hepatocellular jaundice
Non of the above
Which of the following causes is for hepatocellular jaundice
Transfusion reaction
Increased destruction of RBCs
Hepatitis viruses
Non of the above
Obstruction involve small bile ducts within the liver (for long time)
A) hemolytic jaundice
B) hepatocellular jaundice
C) intrahepatic obstruction
D)extrahepatic obstruction
Both B,C
Occlusion of bile duct from a gallstone for long time
A) hemolytic jaundice
B) hepatocellular jaundice
C) intrahepatic obstruction
D) extrahepatic obstruction
Both B,D
A tumor or pressure from an enlarged organ e.g. liver , gallbladder
Hemolytic jaundice
Hepatocellular jaundice
Intrahepatic obstruction
Extrahepatic obstruction
Thickening of bile within the canaliculi after ingestion of certain medication
Hemolytic jaundice
Hepatocellular jaundice
Intrahepatic obstruction
Extrahepatic obstruction
When urine becomes deep orange and foamy that manifestation of
Hemolytic jaundice
Hepatocellular jaundice
Obstructive jaundice
None of the above
Stools become light or clay colored Indicates
Increased amount of bile in the intestinal tract
Decreased amount of bile in the intestinal tract
Presence of blood in stool
High blood glucose
Patient might have liver disease, the nurse draw blood for CBC what will be found to confirm the case's disease
High glucose
Low bilirubin
High GGT in blood
All of the above
Resulting from any of several inherited disorders can also produce jaundice
Hemolytic jaundice
Hereditary hyperbilirubinemia
Hepatocellular jaundice
Obstructive jaundice
Serum bilirubin levels are increased, liver histology and liver function test results are normal, and there is no hemolysis
Hemolytic jaundice
Hepatocellular jaundice
Hereditary hyperbilirubinemia
Obstructive jaundice
The increased pressure throughout the portal venous system that results from obstruction of blood flow
Hereditary hyperbilirubinemia
Ascites
Jaundice
Portal Hypertension
The failure of the liver to metabolize aldosterone increases sodium and water retention
Hereditary hyperbilirubinemia
Ascites
Jaundice
Portal Hypertension
Movement of fluid from the vascular system into the peritoneal space
Hereditary hyperbilirubinemia
Jaundice
Ascites
Portal Hypertension
Distended veins Umbilical hernias are clinical manifestation of
Hereditary hyperbilirubinemia
Ascites
Jaundice
Portal Hypertension
The dietary modification for ascites patient is
High sodium
High fat
Low sodium
Low fat
Which of the following medications is required for ascites patient
Antibiotecs
Diuretics
Topical medicines
Supplements
In patient with ascites a bed rest should be in postion
Lateral posture
Lying
Squatting
Upright posture
Removal of fluid from the peritoneal cavity through a puncture or small surigcal incision
Neurectomy
Paracentesis
Lithotripsy
Nerve biopsy
Used to preform guided liver biopsy and to determine the etiology of ascites
Laparoscopy
MRI
Radioisotope
Paracentesis
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