Clinical Chemistry Carbohydrates

Glucose + Glucose
Fructose
Sucrose
Maltose
Lactose
Glucose + Galactose
Fructose
Sucrose
Maltose
Lactose
Sucrose breaks down into?
Glucose + Galactose
Glucose + Glucose
Fructose + Maltose
Fructose + Glucose
Are the chaining of 2 to 10 sugar units, whereas polysaccharides are formed by the linkage of many monosaccharide units.
MONOSACCHARIDES
DISACCHARIDES
OLIGOSACCHARIDES
POLYSACCHARIDES
Are simple sugars that cannot be hydrolyzed to a simpler form
MONOSACCHARIDES
DISACCHARIDES
OLIGOSACCHARIDES
POLYSACCHARIDES
Formation of glucose-6-phosphate from noncarbohydrate sources.
Glycolysis
Glycogenogenesis
Gluconeogenesis
Glycogenesis
Conversion of carbohydrates to fatty acids
Gluconeogenesis
Lipogenesis
Glycogenogenesis
Lipolysis
Conversion of glucose to glycogen for storage
Gluconeogenesis
Glycogenogenesis
Glucogenesis
Glycogenesis
Metabolism of glucose molecule to pyruvate or lactate for production of energy
Glucogenesis
Glycogenesis
Glycolysis
Glycogenogenesis
The only monosaccharide used by the body for energy
Lipids
Protein
Glucose
Fructose
Is the primary hormone responsible for increasing glucose levels. It is synthesized by the α-cells of islets of Langerhans in the pancreas and released during stress and fasting states.
Somatostatin
Glucagon
Insulin
Epinephrine
Is the primary hormone responsible for the entry of glucose into the cell. It is synthesized by the β-cells of islets of Langerhans in the pancreas.
Somatostatin
Glucagon
Insulin
Epinephrine
produced by the Delta cells of the islets of Langerhans of the pancreas, increases plasma glucose levels by the inhibition of insulin, glucagon, growth hormone, and other endocrine hormones.
Somatostatin
Glucagon
Insulin
Epinephrine
Considered more accurate than the glucose oxidase method
Most specific enzyme reacting with only B-D-glucose.
End product of Folin Wu Test
Phosphomolybdenum blue
Cuprous-neocuproine complex (Blue)
Arsenomolybdenum blue
Cuprous-neocuproine complex (Yellow)
End product of Nelson-Somogyi Test
Phosphomlybdenum blue
Cuprous-neocuproine complex (Blue)
Arsenomolybdenum blue
Cuprous-neocuproine complex (Yellow)
It is used for the detection and quantitation of reducing substances in the body fluids.
Nelson-Somogyi Test
Glucose Inverse Colorimetric method
Benedict's Method
Glucose Oxidase method
Stabilizing agent for Benedict's Method
Glucose Inverse Colorimetric method
Benedict's Method
Nelson-Somogyi Test
Folin Wu Test
Alkaline Ferric Reduction
Is a result of cellular-mediated autoimmune destruction of the β-cells of the pancreas, causing an absolute deficiency of insulin secretion.
Type 1 diabetes
Type 2 diabetes
Gestational Diabetes Mellitus
Hypoglycemia
is characterized by hyperglycemia as a result of an individual's resistance to insulin with an insulin secretory defect.
Type 1 diabetes
Type 2 diabetes
Gestational Diabetes Mellitus
Hypoglycemia
Has been defined as any degree of glucose intolerance with onset or first recognition during pregnancy.
Type 1 diabetes
Type 2 diabetes
Gestational Diabetes Mellitus
Hypoglycemia
Is actually a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action, or both.
It is a congenital deficiency of one of three enzymes involved in the galactose metabolism
This disease is a consequence of inherited deficiencies of enzymes that controls the synthesis or the breakdown of glycogen
Is the most common GSD, associated with hyperlipidemia
Glucose level at refrigerated temperature decreases by ______ per hour
5-7 mg/dl
1-2 mg/dl
1-8 mg/dl
1-3 mg/dl
Glucose renal threshold
160-180 mg/dl
170-180 mg/dl
150-180 mg/dl
190-200 mg/dl
MJ experiencing unexpected weight loss from 75.2 Kg to 53.5 Kg. He started to feel excessive thirst and excessive hunger. He went to the hospital for a check-up. Doctors found out that his fasting blood glucose is 129mg/dl. Doctors confirmed that he has diabetes but they want to know the specific type of diabetes. After some tests, they find out that MJ's pancreas does not make insulin because his immune system attacks his islet cells that produce insulin. What type of diabetes does he have?
Type I Diabetes
Type II Diabetes
Gestational Diabetes
Type III Diabetes
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