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Quizzes > Health & Medicine > Diseases & Conditions

Do I Have a Thyroid Disorder? Take the Quiz Now

Ready to test for thyroid problems? Dive into our thyroid dysfunction quiz and self-test your thyroid health!

Difficulty: Moderate
2-5mins
Learning OutcomesCheat Sheet
Paper art illustration of thyroid gland and quiz checklist on golden yellow background

This quick quiz helps you check for signs of a thyroid disorder using your symptoms, like tiredness, mood changes, or weight shifts. In minutes, you'll see patterns and whether it may be time to talk with a doctor. Want to focus on an overactive thyroid instead? Try our quiz on hyperthyroidism .

Which gland regulates metabolic rate by producing thyroid hormones?
Pancreas
Adrenal gland
Pituitary gland
Thyroid gland
The thyroid gland synthesizes and secretes the hormones thyroxine (T4) and triiodothyronine (T3), which are key regulators of metabolism and energy balance. This gland sits in the neck and influences nearly every organ system. Without proper thyroid function, metabolic rate can slow down or accelerate significantly. For more detail, see .
Which hormone is directly produced by the thyroid gland?
TRH
TSH
T4 (Thyroxine)
Cortisol
The thyroid gland produces the hormones triiodothyronine (T3) and thyroxine (T4), which control the body's metabolic rate. T4 is a prohormone that is converted to the more active T3 in peripheral tissues. TSH is produced by the pituitary and TRH by the hypothalamus, while cortisol comes from the adrenal cortex. See for more information.
What does TSH stand for in thyroid function tests?
Thyroid Stimulating Hormone
Triiodothyronine Surface Hormone
Thyroxine Synthetic Hormone
Thyroid Stimulation Hormone
TSH stands for Thyroid Stimulating Hormone, which is produced by the anterior pituitary and regulates thyroid gland activity. It stimulates the thyroid to produce T3 and T4. Measurement of serum TSH is the most sensitive initial screening test for thyroid dysfunction. For further reading, see .
Which of the following is a common symptom of hypothyroidism?
Heat intolerance
Tremors
Weight loss
Weight gain
Hypothyroidism slows metabolic processes, often leading to weight gain, fatigue, cold intolerance, and dry skin. In contrast, weight loss, heat intolerance, and tremors are more typical of hyperthyroidism. Identifying weight gain alongside other signs helps differentiate between thyroid disorders. For more details, visit .
Which of the following is a common symptom of hyperthyroidism?
Cold intolerance
Dry skin
Weight loss
Bradycardia
Hyperthyroidism increases metabolic rate, often causing unexplained weight loss, heat intolerance, palpitations, and tremors. Cold intolerance and dry skin are more commonly associated with hypothyroidism. Recognizing weight loss with other hypermetabolic signs guides the diagnosis. See for more information.
Which test is commonly used as the first-line screening for thyroid function?
TSH level
Anti-TPO antibody testing
Calcitonin level
T3 uptake
Serum TSH is the most sensitive initial test for evaluating thyroid function and can detect both hypo- and hyperthyroidism. If TSH is abnormal, clinicians typically follow up with free T4 and T3 levels. Antibody tests are used to assess autoimmune thyroid diseases, while calcitonin is relevant in medullary carcinoma. Learn more at .
What is a goiter?
Enlarged lymph node
Hypofunctioning adrenal gland
Pituitary tumor
Enlarged thyroid gland
A goiter refers to any enlargement of the thyroid gland, which may result from iodine deficiency, autoimmune diseases, or nodular thyroid disease. Clinically it presents as a visible or palpable neck swelling. Goiters can be diffuse or nodular, and management depends on the underlying cause. For global perspectives, see .
Which mineral is essential for thyroid hormone synthesis?
Zinc
Calcium
Iron
Iodine
Iodine is a critical component of the thyroid hormones T3 and T4; deficiency leads to goiter and hypothyroidism. Iron and zinc are important trace elements but are not directly incorporated into thyroid hormones. Adequate dietary iodine prevents endemic goiter. See for more details.
Which hormone increases the release of thyroid hormones from the thyroid gland?
CRH
TRH
TSH
ACTH
Thyroid Stimulating Hormone (TSH) from the anterior pituitary directly stimulates the thyroid gland to produce and secrete T3 and T4. TRH from the hypothalamus stimulates TSH release but does not act directly on the thyroid. ACTH and CRH regulate the adrenal axis. For further explanation, see .
Where is the thyroid gland located?
In the abdomen near the pancreas
In the brain near the hypothalamus
In the neck below the larynx
In the chest behind the sternum
The thyroid gland lies in the anterior neck, just below the larynx, and wraps around the trachea. Its location allows for easy palpation during physical examination. It is not found in the chest, brain, or abdomen. Detailed anatomy is available at .
In primary hyperthyroidism, what is the typical TSH level?
Elevated
Normal
Suppressed
Undetectable only in secondary hyperthyroidism
Primary hyperthyroidism involves overproduction of thyroid hormones by the gland itself, leading to negative feedback on the pituitary and suppression of TSH. Secondary hyperthyroidism, such as from a TSH-secreting tumor, would elevate TSH. Normal TSH indicates a euthyroid state. More details at .
Which autoantibodies are most commonly associated with Hashimoto's thyroiditis?
Anti-nuclear antibodies
Anti-insulin antibodies
TSH receptor stimulating antibodies
Anti-thyroid peroxidase (anti-TPO)
Hashimoto's thyroiditis is characterized by autoimmune destruction of the thyroid, most commonly associated with anti-thyroid peroxidase (anti-TPO) antibodies. TSH receptor antibodies are associated with Graves' disease. Anti-insulin and anti-nuclear antibodies are seen in other autoimmune diseases. Read more at .
Which symptom is characteristic of Graves' disease but not typical of other hyperthyroid causes?
Exophthalmos
Heat intolerance
Weight loss
Tachycardia
Graves' ophthalmopathy leads to exophthalmos (bulging eyes) due to autoimmune inflammation of orbital tissues, a feature not seen in other forms of hyperthyroidism. Heat intolerance, tachycardia, and weight loss occur in all hyperthyroid conditions. For more, see .
Which medication is a thionamide used to inhibit thyroid hormone synthesis?
Propranolol
Radioactive iodine
Methimazole
Levothyroxine
Methimazole and propylthiouracil are thionamides that inhibit thyroid peroxidase, blocking iodination of thyroglobulin and synthesis of T3 and T4. Levothyroxine is used for hormone replacement in hypothyroidism. Propranolol is a beta-blocker to manage symptoms. See for more information.
What imaging modality is preferred to evaluate thyroid nodules?
Ultrasound
PET scan
MRI
CT scan
Ultrasound is the first-line imaging modality for assessing thyroid nodules; it provides detailed information about size, composition, and vascularity without radiation exposure. CT and MRI are reserved for large or invasive goiters, while PET is used when malignancy is suspected. Guidelines are available at .
Which description best defines a thyroid nodule?
Autoimmune destruction of thyroid cells
A solid or fluid-filled lump within the thyroid gland
Inflammation of the thyroid capsule
Enlargement of the entire gland without discrete lumps
A thyroid nodule is a distinct lesion within the thyroid that can be solid or cystic. It may or may not affect hormone levels. Diffuse enlargement without discrete nodules is termed goiter. For further reading, visit .
Myxedema is best described as which clinical finding?
Exophthalmos
Tremors
Non-pitting mucopolysaccharide swelling
Pitting edema
Myxedema refers to the deposition of mucopolysaccharides in the skin and other tissues in hypothyroidism, causing a characteristic non-pitting edema. Pitting edema is seen in other conditions like heart failure. Exophthalmos occurs in Graves' disease, and tremors in hyperthyroidism. More at .
Which change in body weight is most indicative of untreated hypothyroidism?
No change
Gradual weight gain
Rapid muscle wasting
Sudden weight loss
Untreated hypothyroidism slows metabolic rate leading to gradual weight gain, often accompanied by fluid retention. Sudden weight loss and muscle wasting are more related to hyperthyroidism or systemic illness. Recognizing gradual weight gain with other signs supports the diagnosis. See .
Which lab pattern indicates primary hypothyroidism?
High TSH, high T4
High TSH, low free T4
Low TSH, low T4
Low TSH, high T4
In primary hypothyroidism, the thyroid gland fails to produce adequate T4, resulting in low free T4 levels. The pituitary responds by increasing TSH secretion, leading to elevated TSH. Low TSH with low T4 suggests central hypothyroidism. For more, see .
What characterizes postpartum thyroiditis?
Permanent hyperthyroidism after childbirth
Permanent hypothyroidism after childbirth
Hyperthyroid then hypothyroid phase after childbirth
Hypothyroid then hyperthyroid phase after childbirth
Postpartum thyroiditis typically presents with a transient hyperthyroid phase shortly after delivery followed by a hypothyroid phase before normalizing. It is an autoimmune inflammation triggered by pregnancy. Most women recover normal thyroid function within a year. For details, see .
What is the mechanism of action of methimazole in treating hyperthyroidism?
Blocks thyroid hormone receptors
Inhibits TSH secretion
Destroys thyroid tissue with radiation
Blocks iodine organification by inhibiting thyroid peroxidase
Methimazole inhibits thyroid peroxidase, the enzyme responsible for iodinating tyrosine residues on thyroglobulin and coupling iodotyrosines to form T3 and T4. This reduces the synthesis of thyroid hormones. It does not affect TSH secretion or destroy tissue directly. More information at .
Which antibody stimulates the TSH receptor in Graves' disease?
Anti-nuclear antibody
Anti-thyroid peroxidase (anti-TPO)
Anti-thyroglobulin
Thyroid-stimulating immunoglobulin (TSI)
In Graves' disease, thyroid-stimulating immunoglobulins (TSI) bind to and activate the TSH receptor, leading to increased thyroid hormone production. Anti-TPO and anti-thyroglobulin antibodies are associated with Hashimoto's. ANA is related to systemic autoimmune diseases. Refer to for more.
What lab findings define subclinical hypothyroidism?
Elevated TSH, normal free T4
Low TSH, normal free T4
Normal TSH, low free T4
Elevated TSH, low free T4
Subclinical hypothyroidism is diagnosed when TSH is elevated but free T4 levels remain within the normal reference range. Patients may be asymptomatic or have mild symptoms. If TSH rises further or symptoms worsen, treatment may be considered. American Thyroid Association guidelines are available at .
Which management approach is recommended for subclinical hyperthyroidism in elderly patients?
High-dose levothyroxine
Immediate thyroidectomy
Beta-blocker therapy only
Observation and periodic laboratory monitoring
Mild subclinical hyperthyroidism in the elderly is often managed with observation and periodic TSH monitoring, as risks of overtreatment exist. Treatment is reserved for TSH <0.1 mIU/L or if atrial fibrillation, osteoporosis, or symptoms develop. Beta-blockers may treat symptoms but do not correct hormone levels. See .
What is the most common cause of goiter worldwide?
Hashimoto's thyroiditis
Graves' disease
Thyroid cancer
Iodine deficiency
Iodine deficiency remains the most common cause of goiter globally, leading to reduced thyroid hormone synthesis and compensatory gland enlargement. In iodine-replete regions, autoimmune diseases like Graves' and Hashimoto's are more frequent causes. Goiter due to malignancy is relatively rare. WHO resources at explain iodine's role.
How does radioactive iodine uptake differ between Graves' disease and thyroiditis?
Low in both conditions
High in thyroiditis, low in Graves' disease
High in Graves' disease, low in thyroiditis
Normal in both conditions
Graves' disease is characterized by increased uptake of radioactive iodine due to overactive hormone synthesis. In thyroiditis, hormone release is due to gland inflammation and leakage, not new synthesis, leading to low uptake. These patterns help differentiate causes of thyrotoxicosis. For guidelines, see .
Which type of thyroid cancer has the best prognosis?
Medullary carcinoma
Papillary carcinoma
Follicular carcinoma
Anaplastic carcinoma
Papillary thyroid carcinoma is the most common thyroid cancer and has an excellent prognosis, with high survival rates when detected early. Anaplastic carcinoma is aggressive with poor outcomes, and medullary and follicular have intermediate prognoses. Early detection and treatment are key. More at .
In T3 toxicosis, which lab pattern is expected?
Low T3, elevated T4
Elevated T3, normal T4, low TSH
Elevated T4, normal T3, low TSH
Normal T3, elevated TSH
T3 toxicosis is characterized by elevated T3 levels while T4 remains within the normal range, accompanied by suppressed TSH due to negative feedback. This pattern indicates early or mild hyperthyroidism where T3 rises first. Accurate diagnosis requires measuring both hormones. See for lab interpretation.
Which thyroid cell type produces calcitonin?
Follicular cells
Parafollicular (C) cells
Chief cells
Endothelial cells
Calcitonin is secreted by the parafollicular cells (also known as C cells) of the thyroid gland, which help regulate calcium homeostasis. Follicular cells produce T3 and T4, while chief cells are found in the parathyroid glands. Endothelial cells line blood vessels. For more, visit .
What characterizes a thyroid storm?
Autoimmune inflammation of thyroid
Mild hyperthyroid symptoms and low fever
Severe hypercatabolic state with fever, tachycardia, and altered mental status
Hypothyroid crisis with bradycardia
Thyroid storm is a life-threatening exacerbation of hyperthyroidism characterized by high fever, severe tachycardia, agitation or altered mental status, and multiorgan dysfunction. It requires immediate medical treatment with beta-blockers, antithyroid drugs, and supportive care. It differs from routine thyrotoxicosis by its severity and systemic involvement. Clinical guidelines are at .
A gain-of-function mutation in the TSH receptor gene most likely leads to which condition?
Papillary thyroid carcinoma
Toxic autonomous adenoma
Hashimoto's thyroiditis
Graves' disease
A gain-of-function mutation in the TSH receptor results in constitutive receptor activation, leading to a toxic adenoma (hot nodule) that autonomously secretes thyroid hormones independent of TSH. Graves' disease is autoimmune and involves TSI antibodies. Hashimoto's and papillary carcinoma have different pathogeneses. See .
Riedel's thyroiditis is characterized by?
Acute bacterial infection of the thyroid
Lymphocytic infiltration with germinal centers
Multinodular enlargement due to hyperplasia
Fibrous replacement of thyroid tissue extending to adjacent structures
Riedel's thyroiditis is a rare condition where fibrous tissue replaces normal thyroid parenchyma and extends to neck structures, mimicking malignancy. It is part of the IgG4-related disease spectrum. It differs from subacute thyroiditis (granulomatous) and autoimmune (lymphocytic) types. More at .
What histopathological feature is seen in subacute (De Quervain's) thyroiditis?
Granulomatous inflammation with giant cells
Papillary nuclear inclusions
Fibrous obliteration of the thyroid gland
Lymphoid follicles with germinal centers
Subacute (De Quervain's) thyroiditis shows granulomatous inflammation with multinucleated giant cells on histology, often following a viral illness. Fibrosis extending beyond the gland is seen in Riedel's thyroiditis, and lymphoid follicles in chronic autoimmune thyroiditis. Papillary nuclear changes indicate papillary carcinoma. See .
Medullary thyroid carcinoma arises from which cell type?
Lymphoid tissue
Adipocytes
Parafollicular (C) cells
Follicular epithelial cells
Medullary thyroid carcinoma originates from the parafollicular C cells of the thyroid, which produce calcitonin. It is associated with MEN2 syndromes and RET proto-oncogene mutations. Follicular cells produce T3 and T4, while lymphoid tissue and adipocytes are not origins of thyroid carcinoma. For more, see .
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Study Outcomes

  1. Identify Common Thyroid Symptoms -

    After completing the quiz, you'll recognize key signs of a thyroid disorder, such as fatigue, weight fluctuations, and mood changes.

  2. Assess Your Personal Risk -

    Evaluate how your symptoms and lifestyle factors may contribute to thyroid dysfunction based on your quiz responses.

  3. Interpret Quiz Results -

    Understand what your score means and whether it suggests a potential thyroid disorder requiring further evaluation.

  4. Decide on Next Steps -

    Learn when to consult a healthcare professional and how to prepare for a thyroid health assessment.

  5. Learn Thyroid Health Fundamentals -

    Gain foundational knowledge of thyroid function, common disorders, and their impact on overall well-being.

  6. Utilize a Thyroid Disorder Self-Test -

    Apply this self-test approach regularly to monitor your thyroid health over time and spot changes early.

Cheat Sheet

  1. Understanding Thyroid Hormone Feedback -

    Grasp the basics of the hypothalamic-pituitary-thyroid axis: the hypothalamus releases TRH, stimulating pituitary TSH release, which in turn drives thyroid T3 and T4 production. This negative feedback loop keeps hormone levels in check, with typical TSH ranges of 0.4 - 4.0 mIU/L and free T4 of 0.8 - 1.8 ng/dL (American Thyroid Association).

  2. Recognizing Key Clinical Symptoms -

    Spot hallmark signs of thyroid dysfunction such as unexplained weight changes, fatigue, hair thinning and heat or cold intolerance. Use the mnemonic "WISH" - Weight change, Irritability, Sleep disturbance, Heat intolerance - to quickly recall common hypo- and hyperthyroid symptoms (Mayo Clinic).

  3. Interpreting Laboratory Values -

    Learn to read TSH, free T4 and free T3 labs: elevated TSH with low free T4 suggests hypothyroidism, while suppressed TSH with high T4/T3 indicates hyperthyroidism. Remember the simple ratio T4/T3 ≈ 3:1 in healthy adults to flag abnormal conversions (Endocrine Society guidelines).

  4. Leveraging Self-Assessment Quizzes -

    Explore trusted online tools like the "do i have a thyroid disorder quiz" or a thyroid dysfunction quiz to track symptom patterns, but beware they're screening aids - not definitive diagnostics. A solid thyroid disorder self-test or thyroid health quiz can prompt you to gather data before visiting a clinician.

  5. Next Steps & Lifestyle Factors -

    Identify when to consult your doctor by noting risk factors like family history, autoimmune tendencies and persistent symptoms from your test for thyroid problems. Boost your thyroid health with iodine-rich foods, selenium supplementation and stress management - remember the "ISLAND" mnemonic: Iodine, Selenium, Lifestyle, Antioxidants, Nutrition, De-stress.

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